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Specifically, it showed that chronic, excessive alcohol consumption leads to a reduction in dopamine release in the NAc, and that the activation of GDNF signalling in the VTA reduces alcohol consumption by normalizing dopamine levels in the NAc

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Furthermore, interference of PELI1 demonstrated that the regulatory effects of PELI1 on PRMT1 and PRMT5 further influenced the differential changes in arginine methylation modifications of CRIP1 (Fig

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