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Notably, NDBs disrupt bacterial membranes instead of targeting specific metabolic pathways, a mechanism less prone to resistance development (Bustamante-Torres et al., 2022

Meanwhile, BOLA3 deficiency can downregulate glycine cleavage system protein H (GCSH), leading to intracellular glycine accumulation, inducing a metabolic shift towards glycolysis and fatty acid oxidation, generating reactive oxygen species, promoting endothelial cell proliferation, survival, and vasoconstriction, and reducing angiogenic capacity (Yu et al., 2019)