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The increased oxidative stress induced by e-cigarettes stimulates the production of reactive oxygen species (ROS) in alveolar macrophages, causing lung epithelial damage and neutrophil influx at the injury site, leading to aggravated inflammation, mucus production, destruction of alveolar cells, and airway obstruction, potentially increasing future cardiovascular risks [53, 54]
Life expectancy estimates for cohorts provide a summary of the lifetime experience of the cohort, and a comparison between an estimate for the population and never smokers, quantifies the harm done by cigarette smoking
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