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The molecular mechanism may be that high UA increases the phosphorylation of insulin receptor substrate 1 (IRS1) and inhibits the phosphorylation of Akt, which was blocked by N-acetyl-L-cysteine ( Figure 1A ) (Zhi et al., 2016)

However, conflicting evidence arose from a 2012 study, which reported no significant change in testosterone levels but noted a significant decrease in sex hormone-binding globulin (SHBG) after quitting smoking

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