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An animal-model based study showed that treatment of rats with l-carnitine against muscular fatigue reduces free-radical mediated oxidative stress by reducing the production of thiobarbituric acid reactive substances, protein carbonyl, creatine kinase and lipid hydroperoxide [26]

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At high nutrient and ATP levels, AMPK activity is inhibited, and mTORC1 phosphorylates ULK1 and ATG13L of the autophagy-activating protein complex and thus suppresses autophagy [267, 268]

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