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The reduction in negativity observed in B-NLPs can be attributed to the negative charge of phosphate groups in the DNAzyme backbone at physiological pH 53

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Further, pharmacological inhibition of mitochondrial enzyme complexes results in decreased oxidative phosphorylation and seizure activity ( There are numerous indications that increased ROS production occurs across epilepsy models, as evidenced by oxidative damage and impaired redox status

74 Notably, the upregulation of ZEB1 inhibited expression of the gemcitabine transporter via ITGA3/ITGB1/31 signaling and c- JNK pathway, which leads to chemoresistance both in vitro and in vivo