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Histopathological findings revealed preservation of neural architecture, with reduced axonal degeneration, diminished neuronal loss, and limited formation of cystic cavities, suggesting an attenuation of secondary injury processes (12)
As noted, BPC 157 therapy acting via NO-system distinctive presentations previously specified (i.e., L-NAME-responsible/L-arginine responsible, L-NAME-non-responsible/L-arginine-responsible) counteracted opposite disturbances (catalepsy (haloperidol) vs
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