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A blockage of SDH activity causes an accumulation of succinate, which then acts as an oncometabolite by activating signaling pathways, DNA hypermethylation, increase of reactive oxygen species (ROS) production, epithelial-to-mesenchymal transition (EMT), hypoxia-inducible factor (HIF)-1 stabilization, protein modification (such as succinylation) to sustain cancer progression (Fig

mTORC1 is activated by glutamine (Gln), arginine (Arg), and Leucine (Leu), and activates protein synthesis by phosphorylation of eIF4E binding protein 1 (4E-BP1) and ribosomal protein S6 kinase 1 (S6K1)

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